A major switch for the Fanconi anemia DNA damage-response pathway

Nat Struct Mol Biol. 2008 Nov;15(11):1128-30. doi: 10.1038/nsmb1108-1128.
No abstract available

Publication types

  • Research Support, N.I.H., Intramural
  • Research Support, Non-U.S. Gov't
  • Comment

MeSH terms

  • Animals
  • Ataxia Telangiectasia Mutated Proteins
  • Cell Cycle Proteins / genetics
  • Cell Cycle Proteins / metabolism
  • DNA Damage*
  • DNA Repair
  • Fanconi Anemia Complementation Group Proteins / genetics
  • Fanconi Anemia Complementation Group Proteins / metabolism*
  • Fanconi Anemia* / genetics
  • Fanconi Anemia* / metabolism
  • Humans
  • Models, Biological
  • Protein Serine-Threonine Kinases / genetics
  • Protein Serine-Threonine Kinases / metabolism
  • Signal Transduction / physiology*

Substances

  • Cell Cycle Proteins
  • FANCI protein, human
  • Fanconi Anemia Complementation Group Proteins
  • ATR protein, human
  • Ataxia Telangiectasia Mutated Proteins
  • Protein Serine-Threonine Kinases