MiR-139-5p inhibits proliferation and promoted apoptosis of human airway smooth muscle cells by downregulating the Brg1 gene

Respir Physiol Neurobiol. 2017 Dec:246:9-16. doi: 10.1016/j.resp.2017.07.004. Epub 2017 Jul 12.

Abstract

MicroRNAs have emerged as critical regulators in the pathogenesis of asthma. However, the role of microRNAs in asthma needs to be further elucidated. In this study, we found that miR-139-5p was greatly decreased in airway smooth muscle (ASM) cells from asthmatic humans as well as ASM cells stimulated with cytokines. Overexpression of miR-139-5p markedly suppressed ASM cell proliferation and promoted cell apoptosis, whereas knockdown of miR-139-5p had the opposite effect. Further study verified that Brg1, a chromatin remodeling factor, was upregulated in ASM cells treated with cytokines and acted as a direct target of miR-139-5p. Ectopic expression of Brg1 partially reversed the effect of miR-139-5p on cell proliferation and apoptosis. Moreover, overexpression of Brg1 restored miR-139-5p-induced downregulation of Akt and p70S6K phosphorylation. Together, these data indicate that miR-139-5p may function as a key regulator of ASM cell proliferation and apoptosis, potentially by targeting the Brg1 gene, and thus suggesting a potential role of miR-139-5p in the pathogenesis of asthma.

Keywords: Asthma; Brg1; Human airway smooth muscle cell; MicroRNAs.

MeSH terms

  • Analysis of Variance
  • Apoptosis / genetics*
  • Asthma / pathology
  • Bronchi / pathology
  • Cell Proliferation / drug effects
  • Cell Proliferation / genetics*
  • Cells, Cultured
  • Cytokines / metabolism
  • Cytokines / pharmacology
  • DNA Helicases / genetics*
  • DNA Helicases / metabolism
  • Down-Regulation / drug effects
  • Down-Regulation / physiology*
  • Flow Cytometry
  • HEK293 Cells
  • Humans
  • MicroRNAs / genetics
  • MicroRNAs / metabolism*
  • Myocytes, Smooth Muscle / drug effects
  • Myocytes, Smooth Muscle / pathology*
  • Myocytes, Smooth Muscle / physiology
  • Nuclear Proteins / genetics*
  • Nuclear Proteins / metabolism
  • Phosphorylation / genetics
  • RNA, Messenger / metabolism
  • Signal Transduction / physiology
  • Transcription Factors / genetics*
  • Transcription Factors / metabolism
  • Transfection

Substances

  • Cytokines
  • MIRN139 microRNA, human
  • MicroRNAs
  • Nuclear Proteins
  • RNA, Messenger
  • Transcription Factors
  • SMARCA4 protein, human
  • DNA Helicases