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Accession: PRJNA768844 ID: 768844

Multi-omics approaches reveal TNF-mediated inhibition of tissue reparative macrophages is via a gene-selective transcriptional mechanism

This SuperSeries is composed of the SubSeries listed below. Overall design: Refer to individual Series
AccessionPRJNA768844; GEO: GSE185380
TypeUmbrella project
PublicationsDichtl S et al., "Gene-selective transcription promotes the inhibition of tissue reparative macrophages by TNF.", Life Sci Alliance, 2022 Apr;5(4)
SubmissionRegistration date: 5-Oct-2021
computational Biology, Max-Planck-Institute for biochemistry
RelevanceSuperseries
Project Data:
Resource NameNumber
of Links
Sequence data
SRA Experiments45
Publications
PubMed1
PMC1
Other datasets
BioSample1573
GEO DataSets4
GEO Data Details
ParameterValue
Data volume, Supplementary Mbytes1375
SRA Data Details
ParameterValue
Data volume, Gbases227
Data volume, Mbytes78946
Multi-omics approaches reveal TNF-mediated inhibition of tissue reparative macrophages is via a gene-selective transcriptional mechanism encompasses the following 3 sub-projects:
Project TypeNumber of Projects
Epigenomics1
BioProject
accession
OrganismTitle
PRJNA768838Mus musculusMulti-omics approaches reveal TNF-mediated inhibition of tissue reparative macrophages is via a gene-selective transcriptional mechanism (ATAC-Seq) (computational Biology, Max-Planck-Institute...)
Transcriptome or Gene expression2
BioProject
accession
OrganismTitle
PRJNA768860Mus musculusMulti-omics approaches reveal TNF-mediated inhibition of tissue reparative macrophages is via a gene-selective transcriptional mechanism (bulk RNA-Seq) (computational Biology, Max-Planck-Institute...)
PRJNA768861Mus musculusMulti-omics approaches reveal TNF-mediated inhibition of tissue reparative macrophages is via a gene-selective transcriptional mechanism (scRNA-Seq) (computational Biology, Max-Planck-Institute...)

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