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Accession: PRJNA218219 ID: 218219

Ablation of coactivator Med1 regulates bulge keratinocyte stem cells and accelerates epidermal regeneration after injury.

This SuperSeries is composed of the SubSeries listed below. Overall design: Refer to individual Series
AccessionPRJNA218219; GEO: GSE50673
TypeUmbrella project
PublicationsOda Y et al., "Deletion of Mediator 1 suppresses TGFβ signaling leading to changes in epidermal lineages and regeneration.", PLoS One, 2020;15(8):e0238076
SubmissionRegistration date: 6-Sep-2013
Endocrine Research, VA Medical Center UCSF
RelevanceSuperseries
Project Data:
Resource NameNumber
of Links
Publications
PubMed1
PMC1
Other datasets
GEO DataSets4
GEO Data Details
ParameterValue
Data volume, Spots462546
Data volume, Processed Mbytes12
Data volume, Supplementary Mbytes3
Ablation of coactivator Med1 regulates bulge keratinocyte stem cells and accelerates epidermal regeneration after injury. encompasses the following 3 sub-projects:
Project TypeNumber of Projects
Transcriptome or Gene expression3
BioProject
accession
OrganismTitle
PRJNA218221Mus musculusAblation of coactivator Med1 regulates bulge keratinocyte stem cells and accelerates epidermal regeneration after injury [10wk skin] (Endocrine Research, VA Medical...)
PRJNA218222Mus musculusAblation of coactivator Med1 regulates bulge keratinocyte stem cells and accelerates epidermal regeneration after injury [skin wound] (Endocrine Research, VA Medical...)
PRJNA218223Mus musculusAblation of coactivator Med1 regulates bulge keratinocyte stem cells and accelerates epidermal regeneration after injury [10wk keratinocyte] (Endocrine Research, VA Medical...)

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