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Items: 4

1.

Transcriptional effect of various iron treatments on Caco-2 cells.

(Submitter supplied) We investigated transcriptional response of CaCo-2 cells to iron treatments, we studied hemin effect by adding hemin to DMEM-FBS medium and iron deficiency effects in using an iron free medium compared to the same supplemented with FAC (ferric ammonium citrate). Keywords: various iron treatment, differential gene expression, hemin treatment, iron-free
Organism:
Homo sapiens
Type:
Expression profiling by array
Dataset:
GDS2230
Platform:
GPL887
12 Samples
Download data
Series
Accession:
GSE3573
ID:
200003573
2.
Full record GDS2230

Intestinal absorptive cell model response to iron deficiency and overload

Analysis of intestinal CaCo-2 cell line grown in medium free of iron or supplemented with hemin to mimic iron overload. Intestinal iron absorption is crucial for systemic iron homeostasis. Results provide insight into molecular mechanisms regulating iron metabolism.
Organism:
Homo sapiens
Type:
Expression profiling by array, log10 ratio, 4 agent, 2 growth protocol sets
Platform:
GPL887
Series:
GSE3573
12 Samples
Download data
DataSet
Accession:
GDS2230
ID:
2230
3.

Agilent-012097 Human 1A Microarray (V2) G4110B (Feature Number version)

(Submitter supplied) Agilent's Human 1A Oligo Microarray (V2) includes over 20K 60-mer oligonucleotide probes, sourced from the Incyte Foundation Database, RefSeq, and GenBank, and are designed to span conserved exons across the transcripts of the targeted full length genes. Coupled with Agilent's probe selection and validation processes, this design method gives the customer maximum confidence in data quality and prevents redundancy in gene coverage. more...
Organism:
Homo sapiens
8 DataSets
98 Series
1 Related Platform
2172 Samples
Download data: TXT
Platform
Accession:
GPL887
ID:
100000887
4.

CaCo2-DMEM-Hemin_a

Organism:
Homo sapiens
Source name:
Caco-2 cells (channel 1) pool of all used RNA (channel 2)
Platform:
GPL887
Series:
GSE3573
Dataset:
GDS2230
Download data
Sample
Accession:
GSM81964
ID:
300081964
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