Warning: The NCBI web site requires JavaScript to function. more...
An official website of the United States government
The .gov means it's official. Federal government websites often end in .gov or .mil. Before sharing sensitive information, make sure you're on a federal government site.
The site is secure. The https:// ensures that you are connecting to the official website and that any information you provide is encrypted and transmitted securely.
Comparison between gene expression in heart from Emd KO and control mice
PubMed Similar studies Analyze with GEO2R
X-linked Emery-Dreifuss muscular dystrophy model and cardiomyopathy
PubMed Similar studies GEO Profiles Analyze DataSet
Activation of MAPK pathways links LMNA mutations to cardiomyopathy in Emery-Dreifuss muscular dystrophy
PubMed Full text in PMC Similar studies Analyze with GEO2R
Comparison between gene expression in heart from Lmna H222P homozygous and control mice
Comparison between gene expression in heart from Lmna H222P heterozygous and control mice
Lmna H222P homozygous and heterozygous mutant models of Emery-Dreifuss muscular dystrophy: hearts
PubMed Full text in PMC Similar studies GEO Profiles Analyze DataSet
Comparative profiling in 13 muscle disease groups
Various muscle diseases (HG-U133B)
Various muscle diseases (HG-U133A)
EDMD-causing emerin mutant myogenic progenitors exhibit impaired differentiation using similar mechanisms
PubMed Full text in PMC Similar studies SRA Run Selector
transcriptomic data of soleus WT and Lmnap.H222P/H222P pre- and post-symptomatic mice
The nuclear envelope protein Net39 is essential for muscle nuclear integrity and chromatin organization
PubMed Full text in PMC Similar studies
The nuclear envelope protein Net39 is essential for nuclear integrity, chromatin organization, and muscle growth (RNA-Seq)
The nuclear envelope protein Net39 is essential for nuclear integrity, chromatin organization, and muscle growth (ChIP-Seq)
The nuclear envelope protein Net39 is essential for nuclear integrity, chromatin organization, and muscle growth (ATAC-Seq)
Polycomb dysfunctional transcriptional repression in muscle stem cells contributes to lamin dependent muscular dystrophy
Polycomb dysfunctional transcriptional repression in muscle stem cells contributes to lamin dependent muscular dystrophy [ChIP-seq]
Polycomb dysfunctional transcriptional repression in muscle stem cells contributes to lamin dependent muscular dystrophy [RNA-seq]
Expression data from H2K mouse myogenic precursor cells
Loss of a heterochromatin anchor rescues altered genome organization and EDMD muscle defects triggered by a laminopathy mutation
Filters: Manage Filters
Your browsing activity is empty.
Activity recording is turned off.
Turn recording back on