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Striatal gene expression data from 12 months-old Hdh4/Q80 mice and control mice.
PubMed Similar studies Analyze with GEO2R
Mutant huntingtin's effects on striatal gene expression in mice
Striatal gene expression data from 22-month-old CHL2 mice and control mice.
Striatal gene expression data from 12 weeks-old R6/2 mice and control mice
Striatal gene expression data from 12 weeks-old R6/2 mice and control mice (set 2)
Striatal gene expression data from 12 weeks-old R6/2 mice and control mice (set 1)
Striatal gene expression data from 3- and 18-month-old Q92 mice and control mice.
In vivo cell-autonomous transcriptional abnormalities revealed in mice expressing mutant huntingtin in striatal but not cortical neurons
PubMed Full text in PMC Similar studies Analyze with GEO2R
RNA-sequencing of Drosophila expressing mutant Huntingtin in neurons or glia
PubMed Full text in PMC Similar studies SRA Run Selector
Gene expression profiles of striatum and cerebellum from knock-in mouse model of Huntington's disease
Huntington's disease CAG knock-in effect on striatum and cerebellum
PubMed Full text in PMC Similar studies GEO Profiles Analyze DataSet
N17 Modifies Mutant Huntingtin Nuclear Pathogenesis and Severity of Disease in HD BAC Transgenic Mice
Striatal gene expression profile of the YAC 128 mouse
Effect of IL-6 deficiency on gene expression in cell types of control and HD mouse striatum
Truncation of mutant huntingtin in knock-in mice via CRISPR-Cas9 uncovers exon1 huntingtin as a key pathogenic form
Dense time series gene expression data from striatum of Huntington’s disease CAG knock-in mice across multiple genetic backgrounds
Selected Spt5-Pol II inhibitors effectively relieve Huntington’s disease neuropathology and slow down its progression in mice
PubMed Full text in PMC Similar studies
Selected Spt5-Pol II inhibitors effectively relieve Huntington’s disease neuropathology and slow down its progression in mice [INCPMPM-17698]
Selected Spt5-Pol II inhibitors effectively relieve Huntington’s disease neuropathology and slow down its progression in mice [INCPMPM-16068]
The CNS-Heart Axis is a Source of Cardiac Dysfunction in Mouse Models of Huntington’s Disease
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