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Gene Profiling in the Livers of Wild-Type and PPARalpha-Null Mice Exposed to Perfluorooctanoic Acid (PFOA)
PubMed Similar studies Analyze with GEO2R
Gene expression profiling in the lung and liver of low and high dose Perfluorooctanoic Acid exposed mouse fetuses
Low doses of perfluorooctanoic acid effect on fetal liver and lung
PubMed Similar studies GEO Profiles Analyze DataSet
High doses of perfluorooctanoic acid effect on fetal liver and lung
Transcriptional response to PFOA in wild-type and PPARalpha-null mice
Perfluorooctanoic acid effect on livers lacking PPAR-alpha
The effects of perfluorooctanoate on high fat diet induced non-alcoholic fatty liver disease in mice
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Expression data from wild-type and PPARalpha-null mice exposed to perfluorooctane sulfonate (PFOS)
PubMed Full text in PMC Similar studies Analyze with GEO2R
Detailed transcriptomics analysis of the effect of dietary fatty acids on gene regulation in the murine heart [superseries]
Detailed transcriptomics analysis of the effect of the PPARalpha agonist Wy14,643 on gene regulation in the murine heart
Detailed transcriptomics analysis of the effect of dietary fatty acids on gene regulation in the murine heart.
Analysis of the Heat Shock Response in Mouse Liver Reveals Transcriptional Dependence on the Nuclear Receptor PPARα
Transcriptional profilling of liver from C57BL/6 wildtype and PPARα-knockout mice treated with PFOA and GenX after 20 weeks of high-fat diet
PubMed Full text in PMC Similar studies
Gene expression changes in liver tissues from alcohol treated PPARa-null mice
PPARalpha-mediated effects of dietary lipids on intestinal barrier gene expression
Validation of a genomics-based hypothetical adverse outcome pathway: 2,4-dinitrotoluene perturbs PPAR signaling impairing energy metabolism and decreasing exercise endurance
Real-time quantitative PCR analysis of fatty acid metabolism genes of liver tissue from wild-type and PPARα (-/-) mice exposed to 2,4-dinitrotoluene (glucose)
Real-time quantitative PCR analysis of fatty acid metabolism genes of liver tissue from wild-type and PPARα (-/-) mice exposed to 2,4-dinitrotoluene (fatty acid)
Real-time quantitative PCR analysis of fatty acid metabolism genes of liver tissue from wild-type and PPARa (-/-) mice exposed to 2,4-dinitrotoluene (PPARa)
PPARa siRNA-Treated Expression Profiles Uncover the Causal Sufficiency Network for Compound-Induced Liver Hypertrophy
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