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Transcriptional profiling reveals divergent roles of PPARa and PPARß/d in regulation of gene expression in mouse liver
PubMed Full text in PMC Similar studies Analyze with GEO2R
mRNA profiling reveals divergent roles of PPARa and PPARß/d in regulating mouse liver gene expression (PPARb/d samples)
PubMed Similar studies Analyze with GEO2R
mRNA profiling reveals divergent roles of PPARa and PPARß/d in regulating mouse liver gene expression (PPARa samples)
ALD-PPARβ/δ
Gene expression of wild-type and Ppar-beta null primary keratinocytes, with and without infection with an activated Hras retrovirus, with and without the Ppar-beta specific ligand GW0742
Microarray skeletal muscle PPARbeta overexpressing mice
Microarray skeletal muscle PPARalpha overexpressing mice
PPARalpha overexpression effect on skeletal muscles
PubMed Full text in PMC Similar studies GEO Profiles Analyze DataSet
DEHP activation of PPAR(alpha) and CAR regulartory pathway in mouse liver
Peroxisome proliferator-activated receptor alpha deficiency effect on phthalate-exposed liver
Transcriptional profiling of PPARα-/- and CREB3L3-/- livers reveals disparate regulation of hepatoproliferative and metabolic functions of PPARα
Expression profiling of pancreatic beta-cells harboring a pancreatic-specific deletion of PPAR-beta
Pancreatic-specific PPAR-beta deletion effect on pancreatic beta-cells
Porcine liver
UIUC Porcine muscle plus
PubMed Similar studies
PPARalpha-mediated effects of dietary lipids on intestinal barrier gene expression
Analysis of the Heat Shock Response in Mouse Liver Reveals Transcriptional Dependence on the Nuclear Receptor PPARα
Model steatogenic compounds (amiodarone, valproic acid, and tetracycline) alter lipid metabolism by different mechanisms in mouse liver slices
Model steatogenic compounds (amiodarone, valproic acid, and tetracycline) alter lipid metabolism by different mechanisms in mouse liver slices [Cholestatic compounds exposures]
Model steatogenic compounds (amiodarone, valproic acid, and tetracycline) alter lipid metabolism by different mechanisms in mouse liver slices [Steatogenic compounds exposures]
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