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Transcriptional plasticity promotes primary and acquired resistance to BET bromodomain inhibition
PubMed Full text in PMC Similar studies SRA Run Selector
iBET resistance
PubMed Full text in PMC Similar studies Analyze with GEO2R
ChIP of BET proteins in iBET resistance [ChIP-seq]
Genome wide expression analysis of BET inhibitor resistance [RNA-seq]
Genome wide expression analysis of BET inhibitor resistance
Response and resistance to BET bromodomain inhibitors in triple negative breast cancer
PubMed Full text in PMC Similar studies
Response and resistance to BET bromodomain inhibitors in triple negative breast cancer [RNA-Seq]
PubMed Full text in PMC Similar studies Analyze with GEO2RSRA Run Selector
Response and resistance to BET bromodomain inhibitors in triple negative breast cancer [ChIP-Seq]
Response and resistance to BET bromodomain inhibitors in triple negative breast cancer [Chem-Seq]
RNAi screen identifies Brd4 as a therapeutic target in acute myeloid leukemia
RNAPol2 accounts for tumor cells liability to JQ1
RNAPol2 accounts for tumor cells liability to JQ1 [ChIP-Seq]
RNAPol2 accounts for tumor cells liability to JQ1 [Affymetrix]
BET Bromodomain Inhibition Blocks the Function of a Critical AR-Independent Master Regulator Network in Lethal Prostate Cancer
High Resolution Mapping of RNA Polymerases Identifies Mechanisms of Sensitivity and Resistance to BET Inhibitors in t(8;21) AML
BET inhibition releases the Mediator complex from specific cis elements in acute myeloid leukemia cells (RNA-Seq II)
BET inhibition releases the Mediator complex from specific cis elements in acute myeloid leukemia cells (ChIP-Seq II)
BET inhibition releases the Mediator complex from specific cis elements in acute myeloid leukemia cells
BET inhibition releases the Mediator complex from specific cis elements in acute myeloid leukemia cells (RNA-Seq I)
BET inhibition releases the Mediator complex from specific cis elements in acute myeloid leukemia cells (ChIP-Seq I)
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