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Genome-wide binding and mechanistic analyses of Smchd1 mediated epigenetic regulation
PubMed Full text in PMC Similar studies
Genome-wide binding and mechanistic analyses of Smchd1 mediated epigenetic regulation [ChIP-Seq, MBD-Seq]
PubMed Full text in PMC Similar studies SRA Run Selector
Genome-wide binding and mechanistic analyses of Smchd1 mediated epigenetic regulation [RNA-Seq]
Transcriptome profiling for genes transcriptionally regulated by Smchd1 in lymphoma cell lines.
Genome-wide identification of CTCF and Cohesin binding sites in neonatal mouse brain
Investigating the role of SMCHD1 in regulating clustered protocadherin (Pcdh) and Hox genes
Role of SmcHD1 in the establishment of the epigenetic states required for the maintenance of X chromosome inactivation
PubMed Similar studies
Role of SmcHD1 in the establishment of the epigenetic states required for the maintenance of X chromosome inactivation [ChIP-seq]
PubMed Similar studies SRA Run Selector
Role of SmcHD1 in the establishment of the epigenetic states required for the maintenance of X chromosome inactivation [RNA-seq]
Gene expression analysis upon stable SmcHD1 Knock Down
PubMed Full text in PMC Similar studies Analyze with GEO2R
Roles of Structural maintenance of chromosome flexible domain containing 1 (Smchd1) in early lineage formation and development in mice
CTCF binding profiles in MEF cells
The contribution of cohesin-SA1 to chromatin architecture and gene expression in two murine tissues
SMCHD1 variants induce variegated expression in Facio Scapulo Humeral Dystrophy and Bosma Arhinia and microphtalmia syndrome (Methylation).
SMCHD1 variants induce variegated expression in Facio Scapulo Humeral Dystophy and Bosma Arhinia and microphtalmia syndrome (ChIP-Seq).
Epigenetic Repogramming by an Environmental Carcinogen Through Chromatin Domain Disruption
Epigenetic Repogramming by an Environmental Carcinogen Through Chromatin Domain Disruption [RNA-Seq]
PubMed Full text in PMC Similar studies Analyze with GEO2RSRA Run Selector
Epigenetic Repogramming by an Environmental Carcinogen Through Chromatin Domain Disruption [ChIP-Seq]
In situ Hi-C in Smchd1 wild-type and Smchd1 deleted neural stem cells. [II]
RRBS in Smchd1 control and Smchd1 deleted male and female neural stem cells.
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