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RNAseq of IL-36 stimulated primary human keratinocytes
PubMed Similar studies Analyze with GEO2RSRA Run Selector
Integrative responses to IL-17 and TNF-α in human keratinocytes account for key inflammatory pathogenic circuits in psoriasis
PubMed Similar studies Analyze with GEO2R
Transcriptomic Profiling of Peripheral Edge of Lesions to Elucidate the Pathogenesis of Psoriasis Vulgaris
PubMed Full text in PMC Similar studies Analyze with GEO2RSRA Run Selector
Development of gene expression signature for primary epidermal neonatal keratinocytes treated with mature IL-36B cytokine
Keratinocytes contribute intrinsically to psoriasis upon loss of Tnip1 function
PubMed Full text in PMC Similar studies SRA Run Selector
Induction of alternative proinflammatory cytokines accounts for sustained psoriasiform skin inflammation in IL-17C+IL-6KO mice
PubMed Full text in PMC Similar studies Analyze with GEO2R
Induction of alternative proinflammatory cytokines accounts for sustained psoriasiform skin inflammation in IL-17C+IL-6KO mice [array]
Induction of alternative proinflammatory cytokines accounts for sustained psoriasiform skin inflammation in IL-17C+IL-6KO mice [RNA-Seq]
Gene expression profiling in psoriatic lesional and non-lesional skin [brodalumab treatment]
Psoriasis response to brodalumab: dose response and time course
PubMed Similar studies GEO Profiles Analyze DataSet
IL-17A is an essential cytokine to sustain pathogenic cell activation and inflammatory gene circuits in psoriasis vulgaris
Anti-IL-17 monclonal antibody ixekizumab effect on chronic psoriasis
PubMed Full text in PMC Similar studies GEO Profiles Analyze DataSet
Gene Expression Analyses of Homo sapiens Inflammatory Skin Diseases
Expression data from IMQ-induced psoriasis-like skin inflammation in miR-146a-/- and C57BL6J mice
Transcriptome analysis of Psoriasis
Th17 cytokines interleukin (IL)-17 and IL-22 modulate distinct inflammatory and keratinocyte-response pathways
TWEAK mediates inflammation in experimental atopic dermatitis and psoriasis
RNA-seq analysis of IL-1B and IL-36 responses in epidermal keratinocytes identifies a shared MyD88-dependent gene signature
Exploring Molecular Determinants of Disease Progression in Psoriasis by Comparing Different Clinical Subtypes Having Similar Core Transcriptomes
PubMed Full text in PMC Similar studies
Using single-cell transcriptomics to characterise early mechanisms of psoriasis resolution
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