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Notch signalling mediates secondary senescence
PubMed Full text in PMC Similar studies SRA Run Selector
Unbiased identification of CSDE1-regulated targets during oncogene-induced senescence
PubMed Similar studies
Unbiased identification of CSDE1-regulated targets during oncogene-induced senescence [RNA-Seq-sh]
PubMed Similar studies SRA Run Selector
Unbiased identification of CSDE1-regulated targets during oncogene-induced senescence [RNA-Seq]
Unbiased identification of CSDE1-regulated targets during oncogene-induced senescence [polysome profiling]
Unbiased identification of CSDE1-regulated targets during oncogene-induced senescence [i-CLIP]
Expression data from paracrine senescence
PubMed Full text in PMC Similar studies Analyze with GEO2R
Oncogene-induced senescence in primary mouse keratinocytes
MLL1 is essential for the senescence-associated secretory phenotype
PubMed Full text in PMC Similar studies
MLL1 is essential for the senescence-associated secretory phenotype [ChIP-Seq]
MLL1 is essential for the senescence-associated secretory phenotype [RNA-Seq]
PubMed Full text in PMC Similar studies Analyze with GEO2RSRA Run Selector
Wnt-signaling potentiates nevogenesis.
Expression data from 7 Human Melanomas
Expression data from Uninfected and BRAF600V induced melanocytes
Expression data from Control, Uninfected and BRAF infected cells
Unique human beta-cell senescence-associated secretory phenotype (SASP) reveal conserved signaling pathways and heterogeneous factors
SASP signature of beta-cells includes pro-inflammatory and stress response proteins
Senescent beta-cells exhibit a unique secretory phenotype that promotes inflammation and remodeling of the extracellular environment
BRD4 connects enhancer remodeling to senescence immune surveillance
BRD4 connects enhancer remodeling to senescence immune surveillance (RNA-seq)
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