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Collateral DNA damage and NAD+ dependence caused by reactive oxygen species signaling in inflammatory macrophages
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Triacylglycerol synthesis enhances macrophage inflammatory function
NAMPT-mediated NAD+ salvage is essential for mitochondrial function and swelling, muscle fiber integrity and premature survival
Efferocytosis Fuels Requirements of Fatty Acid Oxidation and the Electron Transport Chain to Polarize Macrophages for Tissue Repair
Expression data of peritoneal macrophages from Wild-type mouse cultured in control or serine/glycine-depleted medium.
PubMed Full text in PMC Similar studies Analyze with GEO2R
Oxidative metabolism and PGC-1 attenuate macrophage-mediated inflammation
Sema6D reverse signaling controls lipid metabolism for macrophage polarization linking mTOR to PPARγ
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Transcriptome analysis reveals non-foamy rather than foamy plaque macrophages are pro-inflammatory in atherosclerotic murine models
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Single cell RNA sequencing of aortic CD45+ cells and foam cells from atherosclerotic aorta
Bulk RNA-seq of foamy and non-foamy intimal macrophages from ApoE -/- mouse
Gene expression profile of peroxisome proliferator-activated receptor delta (Ppard)-overexpressing RAW 264.7 macrophages treated vehicle, GW501516 or interleukin-4 (Il-4)
Expression data from human monocyte-derived dendritic cells treated or not with interleukin 17A (IL-17A)
Interleukin 17A effect on monocyte-derived dendritic cells
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