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Genome-wide map of viral transcription factor ORF34 binding in Kaposi's Sarcoma Herpesvirus (KSHV)
PubMed Full text in PMC Similar studies SRA Run Selector
An integrative approach identifies direct targets of the late viral transcription complex and an expanded promoter recognition motif in Kaposi’s sarcoma-associated herpesvirus
PubMed Full text in PMC Similar studies
RNA sequencing analysis to identify gene targets of Kaposi's sarcoma herpesvirus (KSHV) ORF24
K8 CLIP-Seq in KSHV reactivated BCBL-1 cells
Global host gene expression changes in KSHV+ PEL cells upon KSHV reactivation
PubMed Full text in PMC Similar studies Analyze with GEO2RSRA Run Selector
Genome-wide mapping of RTA binding sites in KSHV+ PEL cells during KSHV reactivation.
Virus-like vesicles of Kaposi’s Sarcoma-Associated Herpesvirus activate lytic replication through triggering differentiation signaling
Virus-like vesicles of Kaposi’s Sarcoma-Associated Herpesvirus activate lytic replication through triggering differentiation signaling (microRNA)
Virus-like vesicles of Kaposi’s Sarcoma-Associated Herpesvirus activate lytic replication through triggering differentiation signaling (mRNA)
Transcriptome Analysis of KSHV during de novo primary infection of human B-and endothelial-cells
Transcriptome Analysis of KSHV virions produced from BCBL1 and BAC36 in 293L cells
Transcriptome Analysis of KSHV during de novo primary infection of endothelial (TIVE) cells
Transcriptome Analysis of KSHV during de novo primary infection of human B cells (PBMCs)
Transcriptome Analysis of KSHV during de novo primary infection of human CD14+ cells
Transcriptome Analysis of BAC36 and ORF59 deleted BAC36 during de novo primary infection of human B cells (PBMCs)
RNA decay during gammaherpesvirus infection reduces RNA polymerase II occupancy of host promoters but spares viral promoters
The landscape of transcription initiation across latent and lytic KSHV genomes
Cytomegalovirus Late Transcription Factor Target Sequence Diversity Orchestrates Viral Early to Late Transcription
A herpesvirus protein selectively inhibits cellular mRNA nuclear export
Helminth Infection Reactivates Latent γ-herpesvirus Via Cytokine Competition at a Viral Promoter
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