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Links from GEO DataSets

Items: 5

1.
Full record GDS5879

Pulmonary CDC11c+ cells from young and middle-age animals

Analysis of pulmonary CDC11c+ cells from 6-8 week and 10-13 month old C57BL/6 animals. CDC11c+ cells are key modulators of the immune response in the lung. Results provide insight into molecular mechanisms underlying the decline in immune function associated with aging.
Organism:
Mus musculus
Type:
Expression profiling by array, count, 2 age sets
Platform:
GPL6885
Series:
GSE71868
8 Samples
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2.

Gene expression pattern of pulmonary CD11c+ cells from middle-aged and young mice.

(Submitter supplied) Analysis of function of CD11c+ cells from middle-aged and young mice at gene level. This experiment provided insight into the different genes that plays roles in inflammation, immune response and mainly arachidonic acid cascade that are differentiall expressed in CD11c+ cells from middle aged and young mice.
Organism:
Mus musculus
Type:
Expression profiling by array
Dataset:
GDS5879
Platform:
GPL6885
8 Samples
Download data: TXT
Series
Accession:
GSE71868
ID:
200071868
3.

Host responses contributing to the attenuation of severe acute respiratory syndrome coronaviruses missing E protein domains

(Submitter supplied) Severe acute respiratory syndrome coronavirus (SARS-CoV) causes a respiratory disease leading to death in 10% of the infected people. A mouse adapted SARS-CoV lacking the envelope (E) protein (rSARS-CoV-MA15-ΔE) is attenuated in vivo. To identify E protein domains and host responses that contribute to rSARS-CoV-MA15-ΔE attenuation, several mutants (rSARS-CoV-MA15-E*) containing point mutations or deletions in the amino-terminal or the carboxy-terminal regions of E protein, respectively, were generated. more...
Organism:
Mus musculus
Type:
Expression profiling by array
Platform:
GPL13912
15 Samples
Download data: TXT
Series
Accession:
GSE59185
ID:
200059185
4.

Dynamic Innate Immune Responses of Human Bronchial Epithelial Cells against SARS-CoV and DOHV infection

(Submitter supplied) Severe acute respiratory syndrome-associated coronavirus (SARS-CoV) infection causes an immune-mediated disease. We have recently shown that SARS-CoV-induced epithelial Calu-3 cytokines could exacerbate and dampen host inflammatory and T cell responses, respectively, through modulating the functions of macrophages and dendritic cells, thereby suggesting that not only are lung epithelial cells the primary cells of SARS-CoV infection, but they also involve in initiating and orchestrating the host innate and adaptive immunity. more...
Organism:
Homo sapiens
Type:
Expression profiling by array
Platform:
GPL570
27 Samples
Download data: CEL
Series
Accession:
GSE17400
ID:
200017400
5.

Severe acute respiratory syndrome coronavirus envelope protein regulates cell stress responses and apoptosis

(Submitter supplied) Severe acute respiratory syndrome virus (SARS-CoV) that lacks the envelope (E) gene (rSARS-CoV-ΔE) is attenuated in vivo [1,2]. To identify factors that contribute to rSARS-CoV-ΔE attenuation, gene expression in cells infected by SARS-CoV with or without E gene was compared. Twenty-five stress response genes were preferentially upregulated during infection in the absence of the E gene. In addition, genes involved in signal transduction, transcription, cell metabolism, immunoregulation, inflammation, apoptosis and cell cycle and differentiation were differentially regulated in cells infected with rSARS-CoV with or without the E gene. more...
Organism:
Homo sapiens; Chlorocebus aethiops
Type:
Expression profiling by array
Platform:
GPL570
33 Samples
Download data: CEL
Series
Accession:
GSE30589
ID:
200030589
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