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Full record GDS4200

Cmah knockout effect on mdx model of Duchenne Muscular Dystrophy (DMD): skeletal muscle

Analysis of muscle from mdx males with a human-specific deletion in the Cmah (cytidine monophosphate-sialic acid hydroxylase) gene. Cmah-/-mdx mice show increased disease severity that better approximates DMD in humans. Results provide insight into the contributions of glycosylation defects to DMD.
Organism:
Mus musculus
Type:
Expression profiling by array, count, 4 genotype/variation, 2 strain sets
Platform:
GPL1261
Series:
GSE16438
12 Samples
Download data: CEL

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