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    Pvalb parvalbumin [ Mus musculus (house mouse) ]

    Gene ID: 19293, updated on 27-Jul-2021

    GeneRIFs: Gene References Into Functions

    GeneRIFPubMed TitleDate
    Absence of parvalbumin increases mitochondria volume and branching of dendrites in inhibitory Pvalb neurons in vivo: a point of convergence of autism spectrum disorder (ASD) risk gene phenotypes.

    Absence of parvalbumin increases mitochondria volume and branching of dendrites in inhibitory Pvalb neurons in vivo: a point of convergence of autism spectrum disorder (ASD) risk gene phenotypes.
    Janickova L, Rechberger KF, Wey L, Schwaller B., Free PMC Article

    05/29/2021
    Parvalbumin(+) and Npas1(+) Pallidal Neurons Have Distinct Circuit Topology and Function.

    Parvalbumin<sup>+</sup> and Npas1<sup>+</sup> Pallidal Neurons Have Distinct Circuit Topology and Function.
    Pamukcu A, Cui Q, Xenias HS, Berceau BL, Augustine EC, Fan I, Chalasani S, Hantman AW, Lerner TN, Boca SM, Chan CS., Free PMC Article

    01/23/2021
    Differential Coupling of Adult-Born Granule Cells to Parvalbumin and Somatostatin Interneurons.

    Differential Coupling of Adult-Born Granule Cells to Parvalbumin and Somatostatin Interneurons.
    Groisman AI, Yang SM, Schinder AF., Free PMC Article

    01/9/2021
    Arid1b haploinsufficiency in parvalbumin- or somatostatin-expressing interneurons leads to distinct ASD-like and ID-like behavior.

    Arid1b haploinsufficiency in parvalbumin- or somatostatin-expressing interneurons leads to distinct ASD-like and ID-like behavior.
    Smith AL, Jung EM, Jeon BT, Kim WY., Free PMC Article

    12/5/2020
    Prefrontal parvalbumin cells are sensitive to stress and mediate anxiety-related behaviors in female mice.

    Prefrontal parvalbumin cells are sensitive to stress and mediate anxiety-related behaviors in female mice.
    Page CE, Shepard R, Heslin K, Coutellier L., Free PMC Article

    11/21/2020
    GluN2D-mediated excitatory drive onto medial prefrontal cortical PV+ fast-spiking inhibitory interneurons.

    GluN2D-mediated excitatory drive onto medial prefrontal cortical PV+ fast-spiking inhibitory interneurons.
    Garst-Orozco J, Malik R, Lanz TA, Weber ML, Xi H, Arion D, Enwright JF 3rd, Lewis DA, O'Donnell P, Sohal VS, Buhl DL., Free PMC Article

    08/22/2020
    Inducible and reversible silencing of the Pvalb gene in mice: An in vitro and in vivo study.

    Inducible and reversible silencing of the Pvalb gene in mice: An in vitro and in vivo study.
    Filice F, Blum W, Lauber E, Schwaller B.

    08/15/2020
    Data show that conditional loss of the tuberous sclerosis 1 protein (Tsc1) causes a subset of somatostatin (SST)+ cortical interneurons (CINs) to express parvalbumin (PV) and adopt fast-spiking (FS) properties.

    Tsc1 represses parvalbumin expression and fast-spiking properties in somatostatin lineage cortical interneurons.
    Malik R, Pai EL, Rubin AN, Stafford AM, Angara K, Minasi P, Rubenstein JL, Sohal VS, Vogt D., Free PMC Article

    03/21/2020
    Parvalbumin neurons can act solo whereas most of Somatostatin neurons may act in chorus on Pyramidal cells.

    Parvalbumin-expressing interneurons can act solo while somatostatin-expressing interneurons act in chorus in most cases on cortical pyramidal cells.
    Safari MS, Mirnajafi-Zadeh J, Hioki H, Tsumoto T., Free PMC Article

    06/29/2019
    prefrontal cortical parvalbumin and somatostatin expression and cell density increase during adolescence and are modified by BDNF and sex

    Prefrontal cortical parvalbumin and somatostatin expression and cell density increase during adolescence and are modified by BDNF and sex.
    Du X, Serena K, Hwang WJ, Grech AM, Wu YWC, Schroeder A, Hill RA.

    02/2/2019
    Data indicate that fast-spiking interneurons which typically express parvalbumin (PV) show greater firing coherence with CA1 network oscillations.

    Parvalbumin-expressing interneurons coordinate hippocampal network dynamics required for memory consolidation.
    Ognjanovski N, Schaeffer S, Wu J, Mofakham S, Maruyama D, Zochowski M, Aton SJ., Free PMC Article

    12/1/2018
    Martinotti cells in layers II/III of the mouse primary somatosensory cortex are inhibited by both parvalbumin (PV)- and vasoactive intestinal polypeptide (VIP)-expressing cells.

    Parvalbumin- and vasoactive intestinal polypeptide-expressing neocortical interneurons impose differential inhibition on Martinotti cells.
    Walker F, Möck M, Feyerabend M, Guy J, Wagener RJ, Schubert D, Staiger JF, Witte M., Free PMC Article

    09/1/2018
    Synapses from parvalbumin-expressing interneurons onto hippocampal pyramidal neurons are regulated by neuronal firing, signaling through L-type calcium channels.

    Somatostatin and parvalbumin inhibitory synapses onto hippocampal pyramidal neurons are regulated by distinct mechanisms.
    Horn ME, Nicoll RA., Free PMC Article

    08/4/2018
    This study investigated the distribution of Ca2+-binding proteins (CaBPs), such as calbindin D28k, parvalbumin, and calretinin, in the Superior Olivary Complex of the circling mouse on postnatal day 16.

    Immunohistochemical Localization of Calbindin D28k, Parvalbumin, and Calretinin in the Superior Olivary Complex of Circling Mice.
    Lee JK, Maskey D, Kim MJ.

    07/28/2018
    Axotomy Leads to Reduced Calcium Increase and Upregulated Parvalbumin Followed by Decreased Neighboring Microglial Activation.

    Axotomy Leads to Reduced Calcium Increase and Earlier Termination of CCL2 Release in Spinal Motoneurons with Upregulated Parvalbumin Followed by Decreased Neighboring Microglial Activation.
    Paizs M, Patai R, Engelhardt JI, Katarova Z, Obal I, Siklos L.

    05/12/2018
    HIV-1 Tat causes cognitive deficits and selective loss of parvalbumin, somatostatin, and neuronal nitric oxide synthase expressing hippocampal CA1 interneuron subpopulations.

    HIV-1 Tat causes cognitive deficits and selective loss of parvalbumin, somatostatin, and neuronal nitric oxide synthase expressing hippocampal CA1 interneuron subpopulations.
    Marks WD, Paris JJ, Schier CJ, Denton MD, Fitting S, McQuiston AR, Knapp PE, Hauser KF., Free PMC Article

    01/27/2018
    "reduction of PV(+) neurons" was in all cases, i.e., in PV+/-, Shank1-/- and Shank3B-/- mice, was due to a reduction in Pvalb mRNA and PV protein

    Reduction in parvalbumin expression not loss of the parvalbumin-expressing GABA interneuron subpopulation in genetic parvalbumin and shank mouse models of autism.
    Filice F, Vörckel KJ, Sungur AÖ, Wöhr M, Schwaller B., Free PMC Article

    10/22/2016
    The calcium-binding protein parvalbumin (PV) is a crucial marker in defining the most predominant interneuron subtype within the cerebral cortex.

    NPY+-, but not PV+- GABAergic neurons mediated long-range inhibition from infra- to prelimbic cortex.
    Saffari R, Teng Z, Zhang M, Kravchenko M, Hohoff C, Ambrée O, Zhang W., Free PMC Article

    10/22/2016
    dysfunction of parvalbumin positive interneurons signaling in the prefrontal cortex (PFC) specifically produces deficits in the cognitive domain, but does not give rise to PFC-dependent correlates of negative or positive symptoms.

    Parvalbumin-positive interneurons of the prefrontal cortex support working memory and cognitive flexibility.
    Murray AJ, Woloszynowska-Fraser MU, Ansel-Bollepalli L, Cole KL, Foggetti A, Crouch B, Riedel G, Wulff P., Free PMC Article

    10/1/2016
    Thus PV+ GP neurons are synaptically positioned to directly coordinate activity between BG input nuclei, the striatum and STN, and thalamic-output from the SNr.

    Globus Pallidus Externus Neurons Expressing parvalbumin Interconnect the Subthalamic Nucleus and Striatal Interneurons.
    Saunders A, Huang KW, Sabatini BL., Free PMC Article

    07/16/2016
    The PTEN autism spectrum disorder alleles are hypomorphic in regulating cell size and the parvalbumin/somatostatin ratio in comparison to WT PTEN.

    The parvalbumin/somatostatin ratio is increased in Pten mutant mice and by human PTEN ASD alleles.
    Vogt D, Cho KKA, Lee AT, Sohal VS, Rubenstein JLR., Free PMC Article

    02/20/2016
    These findings confirm that fate determination of cortical interneurons subgroups is crucially influenced by Shh signaling, and provide a system for the further study of interneuron fate and function.

    Duration of culture and sonic hedgehog signaling differentially specify PV versus SST cortical interneuron fates from embryonic stem cells.
    Tyson JA, Goldberg EM, Maroof AM, Xu Q, Petros TJ, Anderson SA., Free PMC Article

    02/20/2016
    Parvalbumin and Npas1 neurons have different topologies within the basal ganglia.

    Parvalbumin+ Neurons and Npas1+ Neurons Are Distinct Neuron Classes in the Mouse External Globus Pallidus.
    Hernández VM, Hegeman DJ, Cui Q, Kelver DA, Fiske MP, Glajch KE, Pitt JE, Huang TY, Justice NJ, Chan CS., Free PMC Article

    11/28/2015
    Nearly all parvalbumin interneurons express Lynx1 did not detect Lypd6 in this population. Conversely, in somatostatin interneurons Lypd6 was found in a subset localized to deep cortical layers but no somatostatin neurons show detectable levels of Lynx1.

    Cortical parvalbumin and somatostatin GABA neurons express distinct endogenous modulators of nicotinic acetylcholine receptors.
    Demars MP, Morishita H., Free PMC Article

    11/14/2015
    Our data indicate that the injury-triggered up-regulation of PV-expression is mediated by inflammatory cytokines, and promotes the motility and adhesion of ependymal cells.

    De novo expression of parvalbumin in ependymal cells in response to brain injury promotes ependymal remodeling and wound repair.
    Szabolcsi V, Celio MR.

    10/31/2015
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