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Series GSE115319 Query DataSets for GSE115319
Status Public on Sep 11, 2018
Title Coronary artery disease genes SMAD3 and TCF21 promote opposing interactive genetic programs that regulate smooth muscle cell differentiation and disease risk
Organism Homo sapiens
Experiment type Expression profiling by high throughput sequencing
Genome binding/occupancy profiling by high throughput sequencing
Third-party reanalysis
Summary This SuperSeries is composed of the SubSeries listed below.
 
Overall design Refer to individual Series
 
Citation(s) 30307970
Submission date Jun 04, 2018
Last update date May 01, 2023
Contact name Thomas Quertermous
E-mail(s) tomq1@stanford.edu
Phone 650-723-5012
Organization name Stanford University
Department Medicine Cardiology
Lab Quertermous
Street address 300 Pasteur Drive
City Stanford
State/province CA
ZIP/Postal code 94305
Country USA
 
Platforms (2)
GPL20301 Illumina HiSeq 4000 (Homo sapiens)
GPL20795 HiSeq X Ten (Homo sapiens)
Samples (8)
GSM3175516 TQ324-SMAD3
GSM3175517 TQ325-IgG
GSM3175518 HCASMC scrambled siRNA_S17
This SuperSeries is composed of the following SubSeries:
GSE115317 Coronary artery disease genes SMAD3 and TCF21 promote opposing interactive genetic programs that regulate smooth muscle cell differentiation and disease risk [ChIP-seq]
GSE115318 Coronary artery disease genes SMAD3 and TCF21 promote opposing interactive genetic programs that regulate smooth muscle cell differentiation and disease risk [RNA-seq]
Relations
BioProject PRJNA474525

Download family Format
SOFT formatted family file(s) SOFTHelp
MINiML formatted family file(s) MINiMLHelp
Series Matrix File(s) TXTHelp

Supplementary file Size Download File type/resource
GSE115319_RAW.tar 368.4 Mb (http)(custom) TAR (of BW, NARROWPEAK)
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