|
| Status |
Public on May 06, 2020 |
| Title |
ARID1A gene status shapes cancer immune phenotype and affects cancer immunotherapy |
| Organism |
Homo sapiens |
| Experiment type |
Genome binding/occupancy profiling by high throughput sequencing
Expression profiling by high throughput sequencing
|
| Summary |
This SuperSeries is composed of the SubSeries listed below.
|
| |
|
| Overall design |
Refer to individual Series
|
| |
|
| Citation(s) |
32027624 |
| Submission date |
May 29, 2019 |
| Last update date |
May 06, 2020 |
| Contact name |
Jing Li |
| E-mail(s) |
jingdsli@umich.edu
|
| Organization name |
University of Michigan
|
| Department |
Surgery
|
| Street address |
500 S State St,
|
| City |
Ann Arbor, |
| State/province |
MI |
| ZIP/Postal code |
48109 |
| Country |
USA |
| |
|
| Platforms (2) |
| GPL16791 |
Illumina HiSeq 2500 (Homo sapiens) |
| GPL20301 |
Illumina HiSeq 4000 (Homo sapiens) |
|
| Samples (42)
|
|
| This SuperSeries is composed of the following SubSeries: |
| GSE131915 |
ATAC-seq in ARID1A WT or KO OVCA429 Cells after interferron treatment |
| GSE131916 |
RNA-seq in ARID1A WT, KO OVCA429 Cells after IFNγ treatment |
| GSE131917 |
Intersection of ARID1A, EZH2 RNA-seq in OVCA429 cells with IFNγ treatment |
|
| Relations |
| BioProject |
PRJNA545311 |
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