NCBI Logo
GEO Logo
   NCBI > GEO > Accession DisplayHelp Not logged in | LoginHelp
GEO help: Mouse over screen elements for information.
          Go
Series GSE34148 Query DataSets for GSE34148
Status Public on Jun 07, 2012
Title Phosphorylated and Sumoylation-Deficient Progesterone Receptors Drive Proliferative Gene Signatures During Breast Cancer Progression (Illumina gene expression analysis)
Organism Homo sapiens
Experiment type Expression profiling by array
Summary Anlaysis of the differential gene expression between T47D cells expressing wild type (WT) progesterone receptor isoform B (PR) or SUMOylation-deficient PR molecules.
 
Overall design Total RNA obtained from T47D breast cancer cells that express either WT PR-B or mutant PR-B (K388R, SUMO-deficient), treated with or without synthetic PR ligand R5020 for 6 h.
 
Contributor(s) Knutson TP, Fan FD
Citation(s) 22697792
Submission date Dec 05, 2011
Last update date Aug 13, 2018
Contact name Todd P Knutson
E-mail(s) knut0297@umn.edu
Phone 612-626-8911
Organization name University of Minnesota
Department Minnesota Supercomputing Institute
Street address 117 Pleasant St SE
City Minneapolis
State/province MN
ZIP/Postal code 55455
Country USA
 
Platforms (1)
GPL10558 Illumina HumanHT-12 V4.0 expression beadchip
Samples (12)
GSM842888 T47D_PR_null_noR5020_6h_rep1
GSM842889 T47D_PR_null_noR5020_6h_rep2
GSM842890 T47D_PR_null_yesR5020_6h_rep1
This SubSeries is part of SuperSeries:
GSE34149 Phosphorylated and Sumoylation-Deficient Progesterone Receptors Drive Proliferative Gene Signatures During Breast Cancer Progression
Relations
BioProject PRJNA156473

Download family Format
SOFT formatted family file(s) SOFTHelp
MINiML formatted family file(s) MINiMLHelp
Series Matrix File(s) TXTHelp

Supplementary file Size Download File type/resource
GSE34148_RAW.tar 26.2 Mb (http)(custom) TAR
GSE34148_non-normalized.txt.gz 3.6 Mb (ftp)(http) TXT
Processed data included within Sample table

| NLM | NIH | GEO Help | Disclaimer | Accessibility |
NCBI Home NCBI Search NCBI SiteMap