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Series GSE57256 Query DataSets for GSE57256
Status Public on May 03, 2014
Title Expression data from adult lungs isolated from TMEM100-deficient mice
Organism Mus musculus
Experiment type Expression profiling by array
Summary Tmem100 was identified as a downstream gene of BMP9-ALK1 signaling. The signaling has been implicated in hereditary hemorrhagic telangiectasia. This study was designed to identify affected genes in TMEM100-deficiect mice. Tmem100 was inactivated in adult Tmem1002loxP/2loxP;ROSA26+/CreER mice by tamoxifen tereatment. Microarray data was obtained from the lungs isolated from tamoxifen treated control and mutant mice.
Overall design Three control mice (Tmem1002loxP/2loxP) and three mutant mice (Tmem1002loxP/2loxP;ROSA26+/CreER) were treated with tamoxifen (two consecutive intraperitoneal injections, 0.1 mg/g of body weight) to activate CreER recombinase. Total RNAs were isolated from lungs 7 days after tamoxifen injection.
Contributor(s) Lee YJ, Oh S, Moon E
Citation(s) 25538155
Submission date May 02, 2014
Last update date Jun 14, 2018
Contact name Young Jae Lee
Organization name Gachon University
Department Lee Gil Ya cancer and Diabetes Institute
Lab Developmental Genetics
Street address 155, Gaetbeol-ro, Yeonsu-gu
City Incheon
ZIP/Postal code 406-840
Country South Korea
Platforms (1)
GPL6885 Illumina MouseRef-8 v2.0 expression beadchip
Samples (6)
GSM1378008 430: control mouse 1
GSM1378009 431: control mouse 2
GSM1378010 438: control mouse 3
BioProject PRJNA246087

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SOFT formatted family file(s) SOFTHelp
MINiML formatted family file(s) MINiMLHelp
Series Matrix File(s) TXTHelp

Supplementary file Size Download File type/resource
GSE57256_RAW.tar 3.1 Mb (http)(custom) TAR
GSE57256_non-normalized.txt.gz 1.0 Mb (ftp)(http) TXT
Raw data are available on Series record
Processed data included within Sample table

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