Dystonia and cerebellar atrophy in Cacna1a null mice lacking P/Q calcium channel activity

FASEB J. 2001 May;15(7):1288-90. doi: 10.1096/fj.00-0562fje.
No abstract available

MeSH terms

  • Animals
  • Calcium Channel Blockers / pharmacology
  • Calcium Channels / genetics
  • Calcium Channels / metabolism*
  • Calcium Channels, N-Type
  • Calcium Channels, P-Type
  • Calcium Channels, Q-Type
  • Cells, Cultured
  • Cerebellar Ataxia / genetics
  • Cerebellar Ataxia / physiopathology*
  • Cerebellum / cytology
  • Cerebellum / drug effects
  • Cerebellum / pathology*
  • Cerebellum / physiopathology
  • Gene Targeting*
  • Humans
  • Mice
  • Mice, Transgenic
  • Nimodipine / pharmacology
  • Patch-Clamp Techniques
  • omega-Conotoxin GVIA / pharmacology
  • omega-Conotoxins / pharmacology

Substances

  • CACNA1A protein, human
  • Calcium Channel Blockers
  • Calcium Channels
  • Calcium Channels, N-Type
  • Calcium Channels, P-Type
  • Calcium Channels, Q-Type
  • omega-Conotoxins
  • voltage-dependent calcium channel (P-Q type)
  • omega-conotoxin-MVIIC
  • Nimodipine
  • omega-Conotoxin GVIA

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