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Year | Number of Results |
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2004 | 1 |
2006 | 1 |
2007 | 1 |
2024 | 0 |
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Matrilin-3 is dispensable for mouse skeletal growth and development.
Mol Cell Biol. 2004 Feb;24(4):1691-9. doi: 10.1128/MCB.24.4.1691-1699.2004.
Mol Cell Biol. 2004.
PMID: 14749384
Free PMC article.
Functional knockout of the matrilin-3 gene causes premature chondrocyte maturation to hypertrophy and increases bone mineral density and osteoarthritis.
van der Weyden L, Wei L, Luo J, Yang X, Birk DE, Adams DJ, Bradley A, Chen Q.
van der Weyden L, et al.
Am J Pathol. 2006 Aug;169(2):515-27. doi: 10.2353/ajpath.2006.050981.
Am J Pathol. 2006.
PMID: 16877353
Free PMC article.
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Decreased chondrocyte proliferation and dysregulated apoptosis in the cartilage growth plate are key features of a murine model of epiphyseal dysplasia caused by a matn3 mutation.
Leighton MP, Nundlall S, Starborg T, Meadows RS, Suleman F, Knowles L, Wagener R, Thornton DJ, Kadler KE, Boot-Handford RP, Briggs MD.
Leighton MP, et al.
Hum Mol Genet. 2007 Jul 15;16(14):1728-41. doi: 10.1093/hmg/ddm121. Epub 2007 May 21.
Hum Mol Genet. 2007.
PMID: 17517694
Free PMC article.
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