PubMed for id: 286410

Year	Number of Results
1994	1
1999	1
2000	1
2002	1
2003	1
2004	3
2005	3
2006	1
2007	1
2011	2
2014	3
2015	6
2016	2
2017	3
2018	4
2019	3
2020	7
2021	5
2022	3
2023	4
2024	0

1

A novel missense variant in ATP11C is associated with reduced red blood cell phosphatidylserine flippase activity and mild hereditary hemolytic anemia.

van Dijk MJ, van Oirschot BA, Harrison AN, Recktenwald SM, Qiao M, Stommen A, Cloos AS, Vanderroost J, Terrasi R, Dey K, Bos J, Rab MAE, Bogdanova A, Minetti G, Muccioli GG, Tyteca D, Egée S, Kaestner L, Molday RS, van Beers EJ, van Wijk R. van Dijk MJ, et al. Am J Hematol. 2023 Dec;98(12):1877-1887. doi: 10.1002/ajh.27088. Epub 2023 Sep 6. Am J Hematol. 2023. PMID: 37671681

2

Reduction in flippase activity contributes to surface presentation of phosphatidylserine in human senescent erythrocytes.

Seki M, Arashiki N, Takakuwa Y, Nitta K, Nakamura F. Seki M, et al. J Cell Mol Med. 2020 Dec;24(23):13991-14000. doi: 10.1111/jcmm.16010. Epub 2020 Oct 26. J Cell Mol Med. 2020. PMID: 33103382 Free PMC article.

3

Transport Cycle of Plasma Membrane Flippase ATP11C by Cryo-EM.

Nakanishi H, Nishizawa T, Segawa K, Nureki O, Fujiyoshi Y, Nagata S, Abe K. Nakanishi H, et al. Cell Rep. 2020 Sep 29;32(13):108208. doi: 10.1016/j.celrep.2020.108208. Cell Rep. 2020. PMID: 32997992 Free article.

4

ATP11C T418N, a gene mutation causing congenital hemolytic anemia, reduces flippase activity due to improper membrane trafficking.

Arashiki N, Niitsuma K, Seki M, Takakuwa Y, Nakamura F. Arashiki N, et al. Biochem Biophys Res Commun. 2019 Aug 27;516(3):705-712. doi: 10.1016/j.bbrc.2019.06.092. Epub 2019 Jun 25. Biochem Biophys Res Commun. 2019. PMID: 31253392

5

ATP11C is a major flippase in human erythrocytes and its defect causes congenital hemolytic anemia.

Arashiki N, Takakuwa Y, Mohandas N, Hale J, Yoshida K, Ogura H, Utsugisawa T, Ohga S, Miyano S, Ogawa S, Kojima S, Kanno H. Arashiki N, et al. Haematologica. 2016 May;101(5):559-65. doi: 10.3324/haematol.2016.142273. Epub 2016 Mar 4. Haematologica. 2016. PMID: 26944472 Free PMC article.

6

Impaired Hepatic Uptake by Organic Anion-Transporting Polypeptides Is Associated with Hyperbilirubinemia and Hypercholanemia in Atp11c Mutant Mice.

Matsuzaka Y, Hayashi H, Kusuhara H. Matsuzaka Y, et al. Mol Pharmacol. 2015 Dec;88(6):1085-92. doi: 10.1124/mol.115.100578. Epub 2015 Sep 23. Mol Pharmacol. 2015. PMID: 26399598

7

ATP11C promotes the differentiation of pre-B cells into immature B cells but does not affect their IL-7-dependent proliferation.

Yabas M, Bostanci A, Aral S. Yabas M, et al. Immunol Res. 2023 Aug;71(4):609-616. doi: 10.1007/s12026-023-09364-6. Epub 2023 Feb 8. Immunol Res. 2023. PMID: 36753036

8

The ratio of ATP11C/PLSCR1 mRNA transcripts has clinical significance in sickle cell anemia.

Pereira-Martins DA, Coelho-Silva JL, Domingos IF, Weinhäuser I, Franca-Neto PL, Araujo AS, Franca RF, Bezerra MA, Lucena-Araujo AR. Pereira-Martins DA, et al. Ann Hematol. 2022 Feb;101(2):281-287. doi: 10.1007/s00277-021-04696-5. Epub 2021 Oct 14. Ann Hematol. 2022. PMID: 34651249

9

Crystal structure of a human plasma membrane phospholipid flippase.

Nakanishi H, Irie K, Segawa K, Hasegawa K, Fujiyoshi Y, Nagata S, Abe K. Nakanishi H, et al. J Biol Chem. 2020 Jul 24;295(30):10180-10194. doi: 10.1074/jbc.RA120.014144. Epub 2020 Jun 3. J Biol Chem. 2020. PMID: 32493773 Free PMC article.

10

The cytoplasmic C-terminal region of the ATP11C variant determines its localization at the polarized plasma membrane.

Takayama M, Takatsu H, Hamamoto A, Inoue H, Naito T, Nakayama K, Shin HW. Takayama M, et al. J Cell Sci. 2019 Sep 2;132(17):jcs231720. doi: 10.1242/jcs.231720. J Cell Sci. 2019. PMID: 31371488 Free article.

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