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1976 1
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Page 1
The pathogenesis of insulin resistance: integrating signaling pathways and substrate flux.
Samuel VT, Shulman GI. Samuel VT, et al. J Clin Invest. 2016 Jan;126(1):12-22. doi: 10.1172/JCI77812. Epub 2016 Jan 4. J Clin Invest. 2016. PMID: 26727229 Free PMC article. Review.
Macrophage-induced WAT lipolysis also stimulates hepatic gluconeogenesis, promoting fasting and postprandial hyperglycemia through increased fatty acid delivery to the liver, which results in increased hepatic acetyl-CoA content, a potent activator of pyruvate ca …
Macrophage-induced WAT lipolysis also stimulates hepatic gluconeogenesis, promoting fasting and postprandial hyperglycemia through increased …
Effects of dietary fatty acids on insulin sensitivity and secretion.
Manco M, Calvani M, Mingrone G. Manco M, et al. Diabetes Obes Metab. 2004 Nov;6(6):402-13. doi: 10.1111/j.1462-8902.2004.00356.x. Diabetes Obes Metab. 2004. PMID: 15479216 Review.
The depletion of IMTG depots is strictly associated with an improvement of insulin sensitivity, via a reduced acetyl-CoA carboxylase (ACC) mRNA expression and an increased GLUT4 expression and pyruvate dehydrogenase (PDH) activity. ...In rat models, beta-cell …
The depletion of IMTG depots is strictly associated with an improvement of insulin sensitivity, via a reduced acetyl-CoA carboxylase …
Marginal biotin deficiency is teratogenic in mice and perhaps humans: a review of biotin deficiency during human pregnancy and effects of biotin deficiency on gene expression and enzyme activities in mouse dam and fetus.
Mock DM. Mock DM. J Nutr Biochem. 2005 Jul;16(7):435-7. doi: 10.1016/j.jnutbio.2005.03.022. J Nutr Biochem. 2005. PMID: 15992686 Review.
In studies of mice, a similar degree of biotin deficiency induces characteristic fetal malformations at a high rate. Fetal hepatic biotin content and PCC activity decrease indicating that the fetuses also become biotin deficient. Fetal hepatic acetyl-CoA carboxyl …
In studies of mice, a similar degree of biotin deficiency induces characteristic fetal malformations at a high rate. Fetal hepatic biotin co …
Impact of the renin-angiotensin system on cardiac energy metabolism in heart failure.
Mori J, Zhang L, Oudit GY, Lopaschuk GD. Mori J, et al. J Mol Cell Cardiol. 2013 Oct;63:98-106. doi: 10.1016/j.yjmcc.2013.07.010. Epub 2013 Jul 22. J Mol Cell Cardiol. 2013. PMID: 23886814 Review.
Heart failure is accompanied by significant perturbations in cardiac energy metabolism that can both decrease cardiac energy supply and decrease cardiac efficiency. Recent evidence suggests that Ang II might be involved in these perturbations in cardiac energy metab …
Heart failure is accompanied by significant perturbations in cardiac energy metabolism that can both decrease cardiac energy supply a …
Energy metabolism in astrocytes: high rate of oxidative metabolism and spatiotemporal dependence on glycolysis/glycogenolysis.
Hertz L, Peng L, Dienel GA. Hertz L, et al. J Cereb Blood Flow Metab. 2007 Feb;27(2):219-49. doi: 10.1038/sj.jcbfm.9600343. Epub 2006 Jul 12. J Cereb Blood Flow Metab. 2007. PMID: 16835632 Review.
The proportion of cortical oxidative metabolism attributed to astrocytes ( approximately 30%) in in vivo nuclear magnetic resonance (NMR) spectroscopic and autoradiographic studies corresponds to their volume fraction, indicating similar oxidation rates in astrocytes and neurons. …
The proportion of cortical oxidative metabolism attributed to astrocytes ( approximately 30%) in in vivo nuclear magnetic resonance (NMR) sp …
Neurochemical changes in Leigh's disease.
Murphy JV. Murphy JV. J Nutr Sci Vitaminol (Tokyo). 1976 Aug;22 SUPPL:69-73. doi: 10.3177/jnsv.22.supplement_69. J Nutr Sci Vitaminol (Tokyo). 1976. PMID: 185345 Review.
A series of children with Leigh's disease had normal hepatic pyruvate carboxylase activity, increased cerebral thiamine diphosphate, and decreased cerebral thiamine triphosphate. ...
A series of children with Leigh's disease had normal hepatic pyruvate carboxylase activity, increased cerebral thiamine …
Regulation of fat metabolism in skeletal muscle.
Jeukendrup AE. Jeukendrup AE. Ann N Y Acad Sci. 2002 Jun;967:217-35. doi: 10.1111/j.1749-6632.2002.tb04278.x. Ann N Y Acad Sci. 2002. PMID: 12079850 Review.
Briefly, accumulation of acetyl-CoA would result in inhibition of pyruvate dehydrogenase (PDH), accumulation of citrate would inhibit phosphofructokinase (PFK), and accumulation of glucose-6-phosphate (G6P) would reduce hexokinase (HK) activity. ...There are several …
Briefly, accumulation of acetyl-CoA would result in inhibition of pyruvate dehydrogenase (PDH), accumulation of citrate would inhibit …
Adult Reye's syndrome: a review with new evidence for a generalized defect in intramitochondrial enzyme processing.
Van Coster RN, De Vivo DC, Blake D, Lombes A, Barrett R, DiMauro S. Van Coster RN, et al. Neurology. 1991 Nov;41(11):1815-21. doi: 10.1212/wnl.41.11.1815. Neurology. 1991. PMID: 1944914 Review.
Biochemical, immunologic, and molecular studies of liver, brain, and skeletal muscle revealed a non-uniform decrease in several mitochondrial residual enzyme activities in liver and brain. Pyruvate carboxylase activity was negligible. Cross-reac …
Biochemical, immunologic, and molecular studies of liver, brain, and skeletal muscle revealed a non-uniform decrease in several mitoc …