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RNA-seq: Loss of a heterochromatin anchor rescues altered genome organization and EDMD muscle defects triggered by a laminopathy mutation
PubMed Full text in PMC Similar studies SRA Run Selector
H3K9me blocks transcription factor activity in differentiated cells to ensure tissue integrity
PubMed Full text in PMC Similar studies
H3K9me blocks transcription factor activity in differentiated cells to ensure tissue integrity [RNA-seq]
H3K9me blocks transcription factor activity in differentiated cells to ensure tissue integrity [CUT&RUN; ChICseq]
H3K9me blocks transcription factor activity in differentiated cells to ensure tissue integrity [ATAC-seq]
Loss of a heterochromatin anchor rescues altered genome organization and EDMD muscle defects triggered by a laminopathy mutation
DamID: Loss of a heterochromatin anchor rescues altered genome organization and EDMD muscle defects triggered by a laminopathy mutation
Next generation sequencing facilitates quantitative analysis of Caenorhabditis elegans N2 wild type, emr-1(gk119), lem-2(tm1582) and emr-1(RNAi) lem-2(tm1582) transcriptomes.
DamID of Dam::LMN-1 and Dam::EMR-1 in Caenorhabditis elegans adult worms
PubMed Full text in PMC Similar studies Analyze with GEO2R
A structural role for histone methyltransferase MET-2 represses transcription independent of H3K9me catalysis
A structural role for histone methyltransferase MET-2 represses transcription independent of H3K9me catalysis [RNA-seq]
A structural role for histone methyltransferase MET-2 represses transcription independent of H3K9me catalysis [ChIP-seq]
An unstructured MET-2/SETDB1 cofactor ensures H3K9me2, focus formation and perinuclear anchoring
An unstructured MET-2/SETDB1 cofactor ensures H3K9me2, focus formation and perinuclear anchoring [RNA-seq]
An unstructured MET-2/SETDB1 cofactor ensures H3K9me2, focus formation and perinuclear anchoring [ChIP-seq]
Comparative profiling in 13 muscle disease groups
Various muscle diseases (HG-U133B)
PubMed Full text in PMC Similar studies GEO Profiles Analyze DataSet
Various muscle diseases (HG-U133A)
Histone H3K9 methylation promotes formation of genome compartments in C. elegans via chromosome compaction and perinuclear anchoring
Histone H3K9 methylation promotes formation of genome compartments in C. elegans via chromosome compaction and perinuclear anchoring (Hi-C)
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