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Status |
Public on Sep 04, 2019 |
Title |
Transcriptome profiling of 57 LUAD samples and 11 peritumoral normal lung tissues |
Organism |
Homo sapiens |
Experiment type |
Expression profiling by array
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Summary |
RNA was extracted from 57 LUAD patients hospitalized from 2004 to 2010 at the Pasteur Hospital (Departments of Pulmonary Medicine, and Thoracic Surgery, CHU de Nice, France) and 11 normal lung tissue specimens taken from areas at standard distance (3 cm) from the same cohort of patients.The diagnosis of LUAD patients was based on examination of all tumor specimens using the 7th pTNM classification and on the last histological classification of NSCLC. Written informed consent was obtained from participants after explaining the nature of the study, which was approved by the research ethics board of the Nice University hospital and was performed according to the guidelines of the Declaration of Helsinki. The main clinical and pathological data are summarized. Enrollment of patients in our study was conditioned by stringent criteria such as obtained signed consent, availability of resected surgical specimens, good quality RNA and time of follow-up for surviving patients (min 40 months for surviving patients).
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Overall design |
68 independent samples: 57 LUAD (T) and 11 Healthy lungs (N).
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Web link |
http://www.genomique.info:8080/merge/index
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Contributor(s) |
Rezzonico R, Pottier N, Nottet N, Mari B, Barbry P, Lebrigand K |
Citation(s) |
31417181 |
Submission date |
Jul 11, 2018 |
Last update date |
Dec 04, 2019 |
Contact name |
Kevin Lebrigand |
Organization name |
IPMC/CNRS
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Lab |
Functional Genomics Platform of Nice-Sophia-Antipolis, France.
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Street address |
660 route des lucioles
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City |
Valbonne - Sophia-Antipolis |
ZIP/Postal code |
06560 |
Country |
France |
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Platforms (1) |
GPL17077 |
Agilent-039494 SurePrint G3 Human GE v2 8x60K Microarray 039381 (Probe Name version) |
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Samples (68)
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This SubSeries is part of SuperSeries: |
GSE117049 |
The nuclear hypoxia-regulated NLUCAT1 long non-coding RNA contributes to an aggressive phenotype in lung adenocarcinoma through regulation of oxidative stress |
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Relations |
BioProject |
PRJNA480704 |